An ion-exchange mechanism may have existed in the cadmium-sorption process with the synthesized SPGMA. The cadmium uptake by SPGMA was quantitatively evaluated with equilibrium sorption isotherms. To describe the isotherms mathematically, the experimental data of the removal equilibrium were correlated with the Langmuir, Freundlich, Temkin, and Dubinin-Radushkevich (D-R) isotherm models, and the applicability of these isotherm equations to the sorption systems was compared on the basis of the

correlation coefficients. The applicability of the equilibrium isotherm models followed this order: Freundlich > Langmuir > Temkin > D-R. The maximum sorption capacity, determined from the Langmuir isotherm, was 555.55 mg/g at 25 degrees C. Thermodynamic parameters, including changes in the enthalpy, Gibbs free energy, and entropy, were calculated. Positive values for the change in enthalpy and negative SHP099 concentration values for the change in the Gibbs free energy showed the endothermic and spontaneous nature Torin 1 supplier of sorption, respectively. The relatively small value

of the activation energy (32.12 kJ/mol) confirmed that the cadmium-sorption process was diffusion-controlled. (C) 2010 Wiley Periodicals, Inc. J Appl Polym Sci 118: 3111-3122, 2010″
“Viral infections including those due to cytomegalovirus have been associated with accelerated cardiac allograft vasculopathy (CAV) in clinical trials and some animal models. Evidence demonstrating a direct causal relationship between such infections and de novo formation

of coronary vascular lesions is lacking. Heterotopic murine cardiac transplants were performed in a parental to F1 combination in animals lacking both T- and B-lymphocytes (RAG-/-). Coronary vasculopathy developed almost exclusively in the presence of recipient infection with lymphocytic choriomeningitis virus but not in uninfected controls. This process was also dependent upon the presence of natural killer (NK) cells as depletion of NK cells abrogated the process. These data show that a viral infection in its native host, PF-03084014 and not previously implicated in the production of CAV, can contribute to the development of advanced coronary vascular lesions in cardiac allotransplants in mice. These data also suggest that virus-induced CAV can develop via an NK-cell-dependent pathway in the absence of T- and B-lymphocytes.”
“In situ observation of Raman scattering in glassy sanbornite (BaSi(2)O(5)) showing homogeneous nucleation was performed during heating to investigate the structural variation in the glassy/supercooled-liquid phase in the pre- and postnucleation regimes. It was observed that the formation of Q(2) units accelerates around 400 degrees C, which is well below T(g), just after structural relaxation in the Ba-enriched/-cohesive region.